Search results for "Cytopathic effect"

showing 10 items of 15 documents

Autophagy interferes with human cytomegalovirus genome replication, morphogenesis, and progeny release.

2020

Viral infections are often accompanied by the induction of autophagy as an intrinsic cellular defense mechanism. Herpesviruses have developed strategies to evade autophagic degradation and to manipulate autophagy of the host cells to their benefit. Here we addressed the role of macroautophagy/autophagy in human cytomegalovirus replication and for particle morphogenesis. We found that proteins of the autophagy machinery localize to cytoplasmic viral assembly compartments and enveloped virions in the cytoplasm. Surprisingly, the autophagy receptor SQSTM1/p62 was also found to colocalize with HCMV capsids in the nucleus of infected cells. This finding indicates that the autophagy machinery int…

0301 basic medicineHuman cytomegalovirusCytoplasmEpstein-Barr Virus InfectionsvirusesCytomegalovirusBiology03 medical and health sciencesMultiplicity of infectionmedicineXenophagyAutophagyMorphogenesisHumansMolecular BiologyCytopathic effect030102 biochemistry & molecular biologyAutophagyCell BiologyBECN1biochemical phenomena metabolism and nutritionFibroblastsmedicine.diseaseVirus ReleaseCell biology030104 developmental biologyCytomegalovirus InfectionsMAP1LC3AResearch PaperAutophagy
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Lack of evidence of mimivirus replication in human PBMCs

2018

The Acanthamoeba polyphaga mimivirus (APMV) was first isolated during a pneumonia outbreak in Bradford, England, and since its discovery many research groups devoted efforts to understand whether this virus could be associated to human diseases, in particular clinical signs and symptoms of pneumonia. In 2013, we observed cytopathic effect in amoebas (rounding and lysis) inoculated with APMV inoculated PBMCs (peripheral blood mononuclear cell) extracts, and at that point we interpreted those results as mimivirus replication in human PBMCs. Based on these results we decided to further investigate APMV replication in human PBMCs, by transmission electron microscopy (TEM) and qPCR. No viral fac…

0301 basic medicinevirukset030106 microbiologyImmunologymimivirusReal-Time Polymerase Chain ReactionVirus ReplicationMicrobiologyPeripheral blood mononuclear cellVirus03 medical and health sciencesMultiplicity of infectionBacterial ProteinsMicroscopy Electron TransmissionacanthamoebaViral factoryHumansCytopathic effectMimivirusbiologyDNA Helicasesta1182biology.organism_classificationVirologyHelicase GeneAcanthamoeba030104 developmental biologyInfectious DiseasesDNA ViralPBMCsLeukocytes MononuclearMimiviridaeMicrobes and Infection
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Neutralizing antibody and clinical status of human immunodeficiency virus (HIV)-infected individuals

1989

An assay based on inhibition of cytopathic effect of human immunodeficiency virus (HIV) strains in Molt 4 cells was developed to quantitate neutralizing antibodies (NA) in sera of HIV-infected individuals. The assay was specific and gave results comparable to those obtained by the inhibition of immunofluorescence (IFI) and reverse transcriptase (RT) activity. Attempts were made to correlate the presence and the antibody titres with the clinical status of HIV-infected individuals classified according to Walter Reed staging classification scheme. NA titres correlated inversely with the stage of HIV infection: Compared with acquired immunodeficiency syndrome (AIDS) patients, HIV-infected subje…

AdultMaleT-LymphocytesCross ReactionsHIV AntibodiesVirus ReplicationVirusCell LineSerologyCytopathogenic Effect ViralAcquired immunodeficiency syndrome (AIDS)Neutralization TestsVirologymedicineHumansNeutralizing antibodyCytopathic effectAcquired Immunodeficiency SyndromebiologyImmune SeraHIVmedicine.diseaseVirologyTiterInfectious DiseasesImmunologybiology.proteinFemaleViral diseaseAntibodyJournal of Medical Virology
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Loss of surface fibronectin after infection of cultured cells by HSV-1 and 2

1985

Fibronectin is lost from the surface of HSV infected cells during cell rounding. In order to investigate also the fate of fibronectin during the process of HSV-induced cell-fusion, BHK, Vero as well as primary or secondary rabbit kidney cells were infected with HSV-1 strains producing cell-fusion. By immunofluorescence and immunoelectron microscopy a considerable loss of fibronectin after HSV infection could be demonstrated leaving only irregular clumps of fibronectin containing virus particles on the cell surface. Decrease and disarrangement of fibronectin was similar during cell rounding and cell fusion. Loss of Fibronectin was closely connected with the two types of the cytopathic effect…

Cytochalasin BvirusesImmunoelectron microscopyBiologyKidneyVirus ReplicationCell FusionCell membranechemistry.chemical_compoundCricetinaeVirologymedicineAnimalsSimplexvirusProtease InhibitorsCytochalasin BCells CulturedCytopathic effectCell fusionHerpes SimplexGeneral MedicineActin cytoskeletonVirologyFibronectinsFibronectinActin Cytoskeletonmedicine.anatomical_structurechemistryCell culturebiology.proteinRabbitsArchives of Virology
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The long-term cytoskeletal rearrangement induced by rabbit enteropathogenic Escherichia coli is Esp dependent but intimin independent.

1999

Attaching and effacing rabbit enteropathogenic Escherichia coli (REPEC) of the O103 serogroup adhere diffusely on HeLa cells and trigger a slow progressive cytopathic effect (CPE) characterized by the recruitment of vinculin and the assembly of actin stress fibres. In contrast to REPEC O103, the reference human EPEC strain E2348/69 is unable to trigger the CPE. In this study, we have shown first that the fimbrial adhesin AF/R2, which mediates the diffuse adhesion of REPEC O103, was not sufficient to induce the CPE capability upon E2348/69. Non-polar mutants of REPEC O103 for espA, espB, espD and eae were then constructed. The four mutants were unable to induce attaching and effacing lesions…

DNA BacterialMutantMolecular Sequence DataMicrobiologyBacterial AdhesionMicrobiology03 medical and health sciencesBacterial ProteinsEscherichia coliAnimalsHumansEnteropathogenic Escherichia coliCytoskeletonAdhesins BacterialMolecular Biology[SDV.MP] Life Sciences [q-bio]/Microbiology and ParasitologyActinCytoskeleton030304 developmental biologyIntiminCytopathic effect0303 health sciencesAdhesins Escherichia colibiologyBase Sequence030306 microbiologyEscherichia coli ProteinsGenetic Complementation TestREARRANGEMENTbiochemical phenomena metabolism and nutritionVinculinBacterial adhesin[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyGenes Bacterialbiology.proteinRabbitsCarrier ProteinsBacterial Outer Membrane ProteinsHeLa CellsMolecular microbiology
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Replication of HSV-1 in murine peritoneal macrophages: comparison of various virus strains with different properties.

1984

The in vitro replication of eleven different strains of herpes simplex virus type 1 was studied in resident or thioglycollate-stimulated mouse macrophages. The strains of herpes simplex virus differed in the type of cytopathic effect, induction capacity for herpes simplex virus coded thymidine kinase and pathogenicity in the mouse. Herpes simplex virus replicated better in thioglycollate-stimulated macrophages than in resident macrophages. In vitro ageing of macrophages increased their replicative potency. Herpes simplex virus replicated better in macrophages from homozygous bg/bg C57/BL6J mice than in macrophages from their heterozygous littermates. Separation of macrophages on discontinuo…

ErythrocytesvirusesClone (cell biology)Mice Inbred StrainsBiologymedicine.disease_causeVirus ReplicationThymidine KinaseHerpesviridaeVirusMicrobiologychemistry.chemical_compoundMiceCytopathogenic Effect ViralPhagocytosisVirologymedicineMacrophageAnimalsAscitic FluidSimplexvirusCells CulturedCytopathic effectMacrophagesGeneral MedicineMacrophage ActivationVirologyMice Inbred C57BLHerpes simplex viruschemistryThymidine kinaseEnzyme InductionThymidineArchives of virology
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Cell types infected in human cytomegalovirus placentitis identified by immunohistochemical double staining

1993

Chronic villitis is almost always present in intrauterine infection with human cytomegalovirus (HCMV). The inflammatory response to this virus has been described in detail. However, little is known about the types of placental cells that may be infected by HCMV and six cases of HCMV placentitis were thus investigated to identify the vulnerable cell types. Immunohistochemical double staining analyses were performed using antibodies to HCMV immediate early antigens and to specific cellular marker proteins. Fixed connective tissue cells could be demonstrated to be the predominantly infected cell type in each placental tissue. Endothelial cells and macrophages were also found to be infected in …

Human cytomegalovirusPathologymedicine.medical_specialtyPlacenta DiseasesTransplacental transmissionvirusesCongenital cytomegalovirus infectionCytomegalovirusConnective tissuePathology and Forensic MedicineAntigenPregnancymedicineHumansVimentinMacrophageEndotheliumPregnancy Complications InfectiousAntigens ViralMolecular BiologyCytopathic effectbiologyMacrophagesvirus diseasesCell BiologyGeneral Medicinemedicine.diseaseImmunohistochemistryVirologymedicine.anatomical_structureConnective TissueCytomegalovirus Infectionsbiology.proteinFemaleAntibodyVirchows Archiv A Pathological Anatomy and Histopathology
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Variant toxin B and a functional toxin A produced by Clostridium difficile C34.

2001

A particular property of Clostridium difficile strain C34 is an insertion of approximately 2 kb in the tcdA-C34 gene that does not hinder expression of a fully active TcdA-C34 molecule. Intoxication with TcdA-C34 induced an arborized appearance in eukaryotic cells (D-type cytopathic effect); intoxication with TcdB-C34 induced a spindle-like appearance of cells (S-type cytopathic effect). Inactivation of GTPases with purified toxins revealed that Rho, Rac, Cdc42, and Rap are substrates of TcdA-C34. The variant cytotoxin TcdB-C34 inactivated Rho, Rac, Cdc42, Rap, Ral, and R-Ras. Hence, this is the first ‘S-type’ cytotoxin which inactivates both Rho and R-Ras, and is coexpressed with a ‘D-type…

MaleCell SurvivalBacterial ToxinsClostridium difficile toxin AClostridium difficile toxin BGTPaseEnterotoxinCHO CellsBiologymedicine.disease_causeMicrobiologyMicrobiologyEnterotoxinsBacterial ProteinsCricetinaeGeneticsmedicineAnimalsHumansMolecular BiologyCells CulturedCytopathic effectSkinToxinClostridioides difficileCytotoxinsGenetic VariationClostridium difficileMolecular biologyCdc42 GTP-Binding ProteinDNA Transposable ElementsMicroscopy Electron ScanningFEMS microbiology letters
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Identification of enteroviral infection among infants and children admitted to hospital with acute gastroentritis in Ho Chi Minh City, Vietnam

2005

A total of 276 fecal specimens collected from infants and children admitted to hospital with acute gastroenteritis in Ho Chi Minh City, Vietnam from October 2002 to September 2003, were tested for the presence of enteroviruses by RT-PCR and virus isolation. Enteroviruses were detected in 27 patients by RT-PCR corresponding to 9.8%. However, only four enterovirus strains could be isolated by cell culture with two different cell lines CaCo2 and Vero, showing specific cytopathic effect (CPE). The results clearly indicate that RT-PCR is a sensitive, specific assay to investigate the true burden of acute gastroenteritis due to enteroviruses in clinical fecal specimens. In the present study, ente…

MaleEchovirusvirusesmedicine.disease_causeGenetic analysisVirusFecesVirologyEnterovirus InfectionsPrevalenceHumansMedicinePhylogenyFecesEnterovirusCytopathic effectbusiness.industryAge FactorsInfantvirus diseasesVirologyHo chi minhGastroenteritisMolecular analysisInfectious DiseasesVietnamChild PreschoolEnterovirusFemaleSeasonsbusinessJournal of Medical Virology
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Infection of murine hepatocyte cultures by herpes simplex virus (HSV) 1 and 2

1984

A study was undertaken of the interaction between liver cells and Herpes Simplex Virus (HSV) in vitro. Hepatocytes were obtained from HSV-resistant (C57/B16) and from HSV-susceptible (BALB/c, A/J, C3H) mouse strains and cultured according to standard methods. Each culture was infected with several strains of HSV-type 1 or of HSV-type 2, respectively. The multiplicity of infection was 5. The cytopathic effect was evaluated by light- and electron-microscopy. The number of infectious particles was determined using rabbit kidney or Vero cell cultures. All evaluations were made at different time intervals after infection. No difference concerning the replication rate of HSV-1 and 2 in isolated h…

MaleSimplexvirusfood.ingredientHepatitis Viral HumanvirusesBiologymedicine.disease_causeVirusHerpesviridaeMicrobiologyMicefoodMultiplicity of infectionCytopathogenic Effect ViralSpecies SpecificitymedicineAnimalsSimplexvirusCells CulturedCytopathic effectMice Inbred BALB CMice Inbred C3HHepatologyHerpes SimplexVirologyHerpes simplex virusCell cultureVero cellFemaleLiver
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